|
Lindera
|
B01.650.940.800.575.912.250.595.400.524 |
|
|
Spores, Fungal
|
B05.775.710 |
|
|
Actin Capping Proteins
|
D051344 |
[Actin capping proteins are cytoskeletal proteins that bind to the ends of ACTIN FILAMENTS to regulate actin polymerization.
] |
|
Destrin
|
D051345 |
[A member of the actin depolymerizing factors. Its depolymerizing activity is independent of HYDROGEN-ION CONCENTRATION.
] |
|
Contraceptives, Postcoital, Hormonal
|
D27.505.696.875.360.276.310.235 |
|
|
Mobility Limitation
|
D051346 |
[Difficulty in walking from place to place.
] |
|
Choroid Hemorrhage
|
C11.941.160.177 |
|
|
Antigens, CD1d
|
D23.101.100.894.080.500 |
|
|
CapZ Actin Capping Protein
|
D051347 |
[An actin capping protein that binds to the barbed-ends of ACTIN filaments. It is a heterodimer consisting of an alpha and a beta subunit. It regulates actin assembly by stabilizing actin oligomers for elongation. In SKELETAL MUSCLE, CapZ is localized to the Z-disk.
] |
|
Tropomodulin
|
D051348 |
[An actin capping protein that binds to the pointed-end of ACTIN. It functions in the presence of TROPOMYOSIN to inhibit microfilament elongation.
] |
|
Wiskott-Aldrich Syndrome Protein
|
D051316 |
[WASP protein is mutated in WISKOTT-ALDRICH SYNDROME and is expressed primarily in hematopoietic cells. It is the founding member of the WASP protein family and interacts with CDC42 PROTEIN to help regulate ACTIN polymerization.
] |
|
Dolphins
|
B01.050.150.900.649.313.875.267 |
|
|
Recruitment, Neurophysiological
|
G11.561.601.760 |
|
|
Wiskott-Aldrich Syndrome Protein, Neuronal
|
D051317 |
[A member of the Wiskott-Aldrich syndrome protein family that is found at high levels in NERVE CELLS. It interacts with GRB2 ADAPTOR PROTEIN and with CDC42 PROTEIN.
] |
|
Dihydroorotate Oxidase
|
D08.811.682.660.300 |
|
|
Immunoglobulin gamma-Chains
|
D12.776.377.715.548.705.500.380 |
|
|
beta-N-Acetylhexosaminidases
|
D08.811.277.450.483.180 |
|
|
Transportation
|
J01.937 |
|
|
Intraocular Lymphoma
|
C15.604.515.569.417 |
|
|
Cataplexy
|
D002385 |
[A condition characterized by transient weakness or paralysis of somatic musculature triggered by an emotional stimulus or physical exertion. Cataplexy is frequently associated with NARCOLEPSY. During a cataplectic attack, there is a marked reduction in muscle tone similar to the normal physiologic hypotonia that accompanies rapid eye movement sleep (SLEEP, REM). (From Adams et al., Principles of Neurology, 6th ed, p396)
] |
