|
obsolete modulation by host of symbiont type IV pilus-dependent motility
|
GO_0097615 |
[OBSOLETE. The process in which an organism effects a change in the type IV pilus-dependent motility of a symbiont organism (i.e. the controlled movement of a bacterial cell which is dependent on the presence of type IV pili, and which includes social gliding motility and twitching motility). The symbiont is defined as the smaller of the organisms involved in a symbiotic interaction.] |
|
obsolete positive regulation by host of symbiont type IV pilus-dependent motility
|
GO_0097616 |
[OBSOLETE. Any process in which an organism activates, maintains or increases the frequency, rate or extent of the type IV pilus-dependent motility of a symbiont organism (i.e. the controlled movement of a bacterial cell which is dependent on the presence of type IV pili, and which includes social gliding motility and twitching motility). The symbiont is defined as the smaller of the organisms involved in a symbiotic interaction.] |
|
GO_0097617
|
GO_0097617 |
|
|
dinoflagellate sulcal notch
|
GO_0097618 |
[A dinoflagellate sulcus that extends all the way to the posterior end of the cell (also known as antapex). The presence of a sulcal notch makes the dinoflagellate hypocone appear bilobed.] |
|
PTEX complex
|
GO_0097619 |
[A protein complex that acts as a protein trafficking machinery and is responsible for the export of proteins across the parasitophorous (symbiont-containing) vacuolar membrane and into the human host cell. The PTEX complex is located in the vacuole membrane. It is ATP-powered, and comprises heat shock protein 101 (HSP101; a ClpA/B-like ATPase from the AAA+ superfamily, of a type commonly associated with protein translocons), a parasite protein termed PTEX150, and exported protein 2 (EXP2). EXP2 is the potential channel, as it is the membrane-associated component of the core PTEX complex. Two other proteins, PTEX88 and thioredoxin 2 (TRX2), were also identified as PTEX components.] |
|
negative regulation of meiotic spindle elongation
|
GO_1902120 |
[Any process that stops, prevents or reduces the frequency, rate or extent of meiotic spindle elongation.] |
|
negative regulation of meiotic nuclear division
|
GO_0045835 |
[Any process that stops, prevents, or reduces the frequency, rate or extent of meiosis.] |
|
lithocholic acid binding
|
GO_1902121 |
[Binding to lithocholic acid.] |
|
bile acid binding
|
GO_0032052 |
[Binding to a bile acid, a steroid carboxylic acids occurring in bile.] |
|
chenodeoxycholic acid binding
|
GO_1902122 |
[Binding to chenodeoxycholic acid.] |
|
(-)-pinoresinol catabolic process
|
GO_1902123 |
[The chemical reactions and pathways resulting in the breakdown of (-)-pinoresinol.] |
|
(-)-pinoresinol metabolic process
|
GO_1901598 |
[The chemical reactions and pathways involving (-)-pinoresinol.] |
|
obsolete (+)-pinoresinol metabolic process
|
GO_1902124 |
[OBSOLETE. The chemical reactions and pathways involving (+)-pinoresinol.] |
|
(+)-pinoresinol catabolic process
|
GO_1902125 |
[The chemical reactions and pathways resulting in the breakdown of (+)-pinoresinol.] |
|
(+)-pinoresinol biosynthetic process
|
GO_1902126 |
[The chemical reactions and pathways resulting in the formation of (+)-pinoresinol.] |
|
obsolete (-)-lariciresinol metabolic process
|
GO_1902127 |
[OBSOLETE. The chemical reactions and pathways involving (-)-lariciresinol.] |
|
(-)-lariciresinol catabolic process
|
GO_1902128 |
[The chemical reactions and pathways resulting in the breakdown of (-)-lariciresinol.] |
|
(-)-lariciresinol biosynthetic process
|
GO_1902129 |
[The chemical reactions and pathways resulting in the formation of (-)-lariciresinol.] |
|
(R)-mandelate dehydrogenase activity
|
GO_0097620 |
[Catalysis of the reaction: (R)-2-hydroxy-2-phenylacetate + acceptor = phenylglyoxylate + reduced acceptor.] |
|
monoamine oxidase activity
|
GO_0097621 |
[Catalysis of the reaction: RCH2NHR' + H2O + O2 = RCHO + R'NH2 + H2O2.] |